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May 15th 2024 12:00 PM Eastern Daylight Time

Cell Response: launches proprietary synthetic-lethality screening platform, Resistome™, to combat cancer drug resistance.

LONDON--Cell Response Ltd announced today the launch of its flagship synthetic lethality platform, Resistome™, to transform the way cancer drug resistance is overcome.

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Cell Response’s proprietary synthetic-lethality screening platform, Resistome™, rapidly identifies drug-specific resistance mechanisms and evaluates first-in-class drugs and combinations which can overcome drug-specific resistance to extend life for patients, as well as identifying refined biomarker strategies to improve the stratification of patients for treatment.

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Cell Response’s Resistome platform provides Pharmaceutical and Biotech companies with a unique opportunity to capture a growing market for novel first-in-class drugs and combinations which overcome resistance to existing first-in-class drugs.

 

Yoram Altschuler, Founder and CEO said “Over 90% of cancer patient mortality is due to drug resistance. Resistome is a truly ground-breaking approach to rapidly discovering novel solutions to cancer drug resistance. Our synthetic-lethality platform uses to power of a miniaturized screen of 1000s of gene constructs in parallel on a single Resistome ‘chip’ to rapidly and cost-effectively identify the genes and pathways responsible for drug-specific resistance. What would take years and 10s of millions of dollars with conventional technology we can complete in weeks to months at a fraction of the cost, and with far greater precision.”

 

About Cell Response

 

Cell Response is a global leader in the field of cancer drug resistance. Resistome™ represents a paradigm shift for drug discovery and development to identify drug-specific resistance mechanisms, develop and test first-in-class drugs and combinations to combat resistance, stratify patients using refined biomarker strategies, rank cancer subtypes and patient groups for label expansion, as well as develop a deep understanding of a drug’s specific mechanism of action and off-target activity to optimize a drug’s therapeutic window.

 

Contact

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Media enquiries

Jon Waterman-Smith

jonws@cellresponsetx.com

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